October 12, 2024

Staudenmayer's erroneous test conclusion, concerning Multiple Chemical Sensitivity patients

Whenever you scald your tongue on
hot tea, your tongue will still feel the
scalding sensation, even if you drink
cool water shortly after having been
scalded.  Well, there is a very similar
phenomenon in the lives of chemical
sensitivity patients. It's called masking.

The failure to consider this phenome-
non was the fatal error in a 1980s test
that ended up being obsessively used
in the anti-MCS propaganda of retired
psychiatrist, Stephen Barrett, MD.  He
once said that he was the media,  even
though he was never a household name.
It's important to keep in mind that the blatant attack dog of Big Pharma,
namely retired psychiatrist Dr Stephen Barret, MD, never worked in the
field of Occupational & Environmental Medicine.  Therefore, he has no
professional experience with those suffering from Chemical Allergies
and/or Irritant-induced illnesses.  Such conditions include Reactive Air-
ways Dysfunction Syndrome, Occupational Asthma due to Low Weight
Molecular Agents, Irritant-associated Vocal Cord Dysfunction, etc.  In-
cidentally, low-weight molecular agents are more commonly known as
chemicals.
_________________________________________________________

In his attempt to convince mankind that Chemical Sensitivity is merely
a mental illness, the Stephen Barrett who never examined any chemical
sensitivity patient repeatedly cited a "research undertaking" which was
conducted in Denver during the 1980s.  The test is formally titled:

Double-blind provocation chamber challenges in 20 patients presenting 
with "multiple chemical sensitivity."   The article which detailed the re-
search undertaking was published on August 18, 1993.

The research team who conducted that test consisted in psychologist
Herman Staudenmayer (Ph.d),  allergist John Selner (MD), and chem-
ist Martin P. Buhr (Ph.d).  I was told by someone very well known in
the Chemical Sensitivity world that Herman Staudenmayer appeared
as a brooder, to state it politely.

The title of the test is misleading, in that it was not based on standard
challenge testing, such as the methacholine challenge test which mea-
sures changes in  FEV1.   Rather, the Staudenmayer test was subject-
ive testing;  the type of testing that Barrett condemned as invalid.   So,
we see another instance of hypocrisy in the psychiatrist of early retire-
ment.

Incidentally, FEV1 is the measurement of Forced Expiratory Volume
after one second of exhaling.  In addition, pulmonary experts, from my
experience, will not allow severely sensitive people to take the metha-
choline challenge test, in fear that they "might not recover" the ability
to breath.  For example, an Ivy League trained pulmonary expert for-
bid me to take the test.  In the State where I was at the time, the law
only permitted pulmonary specialists to order methacholine testing.    

Background in Brevity

1) The test consisted in 145 occasions where a test subject had sent in-
     to into his/her chamber an injection of air.  The test subject was then
     instructed to discern if whether or not the injected air was accompa-
     nied by a chemical agent.  Each of the twenty test subjects participat-
     in at least one "provocation challenge." 

2) The challenges were divided into two types:

a) active challenges, 
b) sham challenges. 

Eighty-eight of the provocation challenges were categorized as "sham"
challenges, and they were recorded as injections of chemical-free air.
The other fifty-seven challenges were defined as "active" challenges,
each of which was recorded as the injection of chemical-bearing air.

3) The sham challenges came in two forms:

a) clean air injected alone,
b) clean air accompanied by an aromatic agent.

4) The active challenges also came in two forms:

a) the injection of an airborne chemical alone,
b) an airborne chemical accompanied by an aromatic agent.

5) The aromatic agents were called "maskers."

Maskers used in the "Staudenmayer Test" included:

a) anise oil,
b) cinnamon oil,
c) lemon oil,
d) peppermint spirit (10% oil and 1% leaves.)

4) The overall result of the test, as recorded by the research team, goes
     as follows:  "Individually, none of these patients demonstrated a re-
     liable response pattern across a series of challenges."  The conclu-
     sion was that persons diagnosed with Multiple Chemical Sensitivity
     are merely psychologically ill.

The Invalidating Feature of that Test

The maskers that Barrett cited in his anti-MCS propaganda as having
been used in the "Herman Staudenmayer Test" are known triggers of
adverse reactions in susceptible persons.   That is to say, the maskers
were chemical-bearing agents.

Concerning anything aromatic, keep in mind that the AMA, the world-
renown Mayo Clinic, the American Lung Association, and the Ameri-
can Academy of Allergy, Asthma, & Immunology each recognize, in
publicly accessible print, that "strong odors" can be triggers of adverse
upper and/or lower respiratory reactions in susceptible people, simply
because they are strong odors.   This has included anise oil, cinnamon
oil, lemon oil, and peppermint spirit.

The Chemical Ingredients in the Sample List of Maskers
Used in 'the Staudenmayer Test' that were Alleged to be

Chemical-free

Concerning the sample list of maskers used in the "Staudenmayer Test,"
observe the following:

Anise Oil:

- An active ingredient in it is anethole.
- Anethole's chemical composition is C10H12O.
- Its CAS No. is 104-46-1.
- It is a known trigger to those adversely reactive to it.
- In fact, Anethole is known as p-1-propenylanisode.
- It is also known as 1-methoxy-4-(1-propenyl)benzene.
- Thus, anise oil is a chemical-bearing agent.

In all occasions where anise was used as a masker in a clean air inject-
ion, a chemical-bearing agent was being injected into the test subject's
chamber.   Therefore, to have recorded such an injection as one of
chemical-free air was to have recorded a falsehood.

Cinnamon Oil:

Along with being a "strong odor," cinnamon oil is a bearer of aldehyde.
In fact, the naturally occurring trans-cinnamaldehyde unassistedly be-
comes benzaldehyde in the presence of heat.

In as much, to have recorded a cinnamon oil air injection as a chemical-
free one was to have recorded yet another falsehood.  Cinnamon oil is
a chemical-bearing agent.

Lemon Oil:

The most prevalent constituent in lemon oil is the monoterpene, limo-
nene, aka 4-isopropenyl-1-methyl-cyclohexene.   Limonene de-
velops a potent sensitizing capacity when oxidized, and it's a reputed
skin sensitizer.   In addition, a Swedish research undertaking record-
ed the following:   "Bronchial hyperresponsiveness was related to in-
door concentrations of limonene, the most prevalent terpene."  Lemon
oil also includes the same alpha-pinene that was implicated in oil of
turpentine allergy.

Peppermint:

This aromatic agent is the bearer of Methyl Salicylate, and as is shown
below, it is among the salicylate allergy triggers.   It's also the bearer of
the following sensitizing agents:   (a) alpha-pinene, (b) phellandrene,
and (c) limonene.   It's also the bearer of (d) methone, (e)  mentho-
furane, (f) and methyl acetate.

Now, as far as concerns methyl salicylate, Supplement 5 of the Journal
of the American Society of Consultant Pharmacists, 1999 / Vol. 14,
states:

"Of note, methyl salicylate carries the same warnings as oral 
salicylates and has the potential to cause Reye's Syndrome in 
children with flu-like symptoms, as well as adverse reactions in
those with aspirin allergy, asthma, or nasal polyps."

In as much, to record an airborne injection of peppermint spirit as a
chemical-free one, is to record yet another falsehood.

Dephosphorylation

The research team gave no consideration to the the masking of sensi-
tivity responses; a phenomenon attributed to the involvement of Ca2+
calmodulin phosphatase calcineurin and the ensuing dephosphorylation
that it induces.  Phosphorylation is explained in the following text:

http://www.chemicalsensitization.com/2011/01/proposed-mechanism-for-mcs.html

Barrett's Predictable Response to the Test

As is to be expected, in an article written by him, Barrett recommended
that clinical researchers conduct more tests likened to the one conduct-
ed by Staudenmayer and his colleagues; anise oil, cinnamon oil, and all. 

You should be able to conjecture why he advocated Kangaroo Court
research.
_____________________________________________________

October 11, 2024

Avoidance, aka Environmental Control:
Board-certified doctors' orders.

The AMA, the American Academy of Allergy Asthma and Immunology
(the AAAAI), and the American Lung Association (the ALA) have all
acknowledged the following:

They acknowledged the existence of Chemical Sensitivity as it applies
to Asthma and Asthma-like conditions, as well as sinusitis and adverse
skin conditions such as dermatitis and urticaria (rashes.)  This includes

1}  Occupational Asthma due to Low Weight Molecular Agents,
2}  Irritant-associated Vocal Cord Dysfunction.
3} Reactive Airways Dysfunction Syndrome,
4)  Irritant-induced Asthma, .
5} Small Airways Disease.

All three associations have acknowledged that chemical-bearing agents can
trigger asthma attacks in susceptible persons. This includes chemically laden
fragrance products.  In as much everyone on earth needs air to breath.  No one
needs vain and gluttonous amounts of artificial fragrances products.

Each organization advocates the practice of Environmental Control; 
of avoiding airborne agents which trigger one's asthma.   In fact, the
AMA has formally referred to Avoidance as "Control of Factors Con-
tributing to Asthma Severity."   In French medical Literature, avoid-
ance is known as "Strict Eviction."  

Examples of recognized asthma triggers in the chemical category
include:

[A] "NO2" from gas stoves and fireplaces,  fumes from
        kerosene heaters, and volatile organic compounds
        from carpeting, cabinetry, plywood, particle board,
         and fumes from household cleaning products."

[B] "Air pollutants such as tobacco smoke, wood smoke,
        chemicals in the air and ozone"

      "Occupational exposure to vapors, dusts, gases or fumes"

      "Strong Odors or sprays such as perfumes, household clean-
        sers, cooking fumes (especially from frying), paints, or var-
        nishes"

[C] "Perfume, paint, hair spray, or any strong odors or fumes."

http://www.epa.gov/asthma/chemical_irritants.html

http://asthma.about.com/od/asthmatriggers/qt/chemictriggers.htm

http://www.aaaai.org/patients/publicedmat/tips/occupationalasthma.stm

http://www.lungusa.org/healthy-air/home/resources/cleaning-supplies.html

http://www.lungusa.org/lung-disease/asthma/about-asthma/understanding-asthma.html

http://www.lungusa.org/lung-disease/asthma/living-with-asthma/take-control-of-your-asthma/asthma-triggers.html

An American Lung Association already stated:

"Perfume, room deodorizers, cleaning chemicals, paints, and
  talcum powder are examples of triggers that must be avoided
  or kept at very low levels."

The same American Lung Association furthermore states:

"These 'triggers' can set off a reaction in your lungs and   other 
  parts of your body." Now, place an emphasis on "other parts
  of your body," and keep in mind that:

Avoidance also applies to Anaphylaxis, Chemically-induced Hepatitis,
Irritant Rhinitis, Dermatitis, Urticaria (rashes), Irritant-associated Vo-
cal Cord Dysfunction, Reactive Airways Dysfunction Syndrome, etc.

The AMA's Admitting to the Converse Relationship Between 
Pollution Levels and Hospital Admissions Due to Asthma

Five to six thousand people die each year from asthma, in the United
States alone, and one of the highest asthma-related death rates has
been in Harlem, NY, as well as Detroit.   Ever so coincidentally, the
environs of Harlem are venues for NYC waste sites. Concerning this,
the AMA has expressly stated that:

"fluctuations in the levels of air pollution correlate with asth-
 ma symptoms and hospital admissions."   [Report 4 of the AMA's
 Council on Scientific Affairs (A-98)]
===============================================

The segment posted below appears in another Atlantic America article.
This means that it might look familiar to you.  In fact, the information in
quotations, as well as the asthma chart, was provided by the American
Academy of Allergy Asthma and Immunology, in its publican information
literature.  Needless to say, charts can be invaluable for individuals seek-
ing to learn fact. 

The AAAAI's public education material 
on the subject of Occupational Asthma

Concerning the AAAAI that Barrett cited in his campaign to convince
mankind that Environmental Illness is merely a matter of mental illness,
it published an instructional website about Occupational Asthma.  The
AAAAI has already acknowledged that Occupational Asthma can be
caused by a number of chemicals at nontoxic/ambient levels, afflicting
a number of  workers employed in a number of  industries.

     Acrylates  . . . . . . . . . . . . . . . . . . . Adhesive handlers
     Amines  . . . . . . . . . . . . . . . . . . . .  Shellac & lacquar handlers
     Anhydrides . . . . . . . . . . . . . . . . . . Plastic, epoxy resin users
     Chloramine-T . . . . . . . . . . . . . . . . Janitors, cleaning staff
     Dyes . . . . . . . . . . . . . . . . . . .  . . . . Textile workers
     Fluxes   . . . . . . . . . . . . . . . . . . . . . Electronic workers
     Formaldehyde/glutaraldehyde . . .  Hospital staff
     Persulfate  . . . . . . . . . . . . . . . . . . . Hairdressers
     Isocyanates . . . . . . . . . . . . . . . . . . Spray painters, Insulation
                                                               installers; plastic, rubber,
                                                               foam manufactory workers.

         The same public education material of  the AAAAI states:

          "The cause may be allergic or nonallergic in nature,
            and the disease may last for a lengthy period in some  
            workers, even if they are no longer exposed to the
            agents that caused their symptoms
."

           "Inhalation of  some substances in aerosol form can
            directly lead to the accumulation of  naturally oc-
            curring chemicals in the body, such as histamine or 
            acetylcholine within the lung, which in turn lead to 
            asthma."

            "For example, insecticides, used in agricultural work,
            can cause a buildup of  acetylcholine, which causes
            airway muscles to contract, thereby constricting air-
            ways."

           "Allergic occupational asthma can occur in workers
            in the plastic, rubber or resin industries following
            repeated exposure to small chemical molecules in 
            the air."

          "If occupational asthma is not correctly diagnosed
           early, and the worker protected or removed from the
          exposure, permanent lung changes may occur and 
          asthma symptoms may persist even without exposure."

          "Up to 15% of  asthma cases in the United States may
           have job-related factors."

          "Isocyanates are chemicals that are widely used in many 
           industries, including spray painting, insulation installa-
           tion, and in manufacturing plastics, rubber and foam.
           These chemicals can cause asthma in up to 10% of ex-
           posed workers."

The aforementioned illustrates that Chemical Sensitivity, as it applies to
asthma and rhinitis, is acknowledged as valid and authentic by the same
AAAAI that Barrett elected to use, in order to support his assertion that
chemical sensitivity is merely a psychological illness.

Conclusion

Stephen Barrett can mock the diagnostic title, Multiple Chemical Sensi-
tivity, all that he wants to.  It will not take away the fact is that chemical
sensitivity has already been recognized in case specific form.  Nor will
it take away the fact that the sufferers of those case-specific forms of
chemical sensitivity need to avoid the chemicals which exacerbate their
medical conditions.
_________________
The following quote is
directly from the AMA 

Avoidance and AMA (CSA) Report 4 (A-98)

Avoidance is not 'detrimental.'  Nor is it nonsense.  Avoidance is a
medical necessity.  And as it applies to asthma, the AMA has stated:

   Regardless of the efficiency of clinician assessment and pa-
   tient self-monitoring, if the patient's exposure to irritants
   or allergens to which he or she is sensitive is not reduced 
   or eliminated, symptom control and exacerbation rate may
   not improve.  Formerly titled 'Environmental Control,' the
   key points in this area logically include efforts by clinicians
   to pinpoint causative agents and to provide specific advice 
   on how to avoid or reduce exposures to environmental or
   dietary triggers and drugs that may provoke or exacerbate 
   symptoms." AMA Report 4, Council on Scientific Affairs (A-98)
   ============================================

October 10, 2024

The Proposed Mechanism for Multiple (body system) Chemical Sensitivity

It's popularly known as Multiple Chemical  Sensitivity (aka MCS.)
However, the diagnostic title does NOT refer to the phenomenon of
sensitivity to multiple chemicals.  It refers to chemical sensitivity
simultaneously afflicting multiple body systems, and not merely
one of them.

Concerning sensitivity to multiple chemicals, that phenomenon had long
since been proven to be real in cases of asthma, sinusitis, rhinitis, vocal
cord dysfunction, dermatitis, and a medical condition known as Reactive
Airways Dysfunction Syndrome.

The 21st Century's proposed mechanism for MCS identifies two general
categories of chemical sensitivity.  They are Central Chemical Sensitiv-
ity and Peripheral Chemical Sensitivity.  The outline goes as follows:

Central Chemical Sensitivity

This type of chemical sensitivity involves the central nervous system, and
it's triggering point is proposed to be found in chemoreceptor activation
(action potential.)

Specific chemoreceptors, upon their activation, elevate nitric oxide levels
in the body.  The nitric oxide reacts with superoxide, producing peroxy-
nitrite.

While the nitric oxide is engaged in producing peroxynitrite, it is simul-
taneously engaged in an additional function.  This function is "retrograde
signaling."

Nitric oxide's role in retrograde signaling is proposed to be that of send-
ing an electrical signal to the presynapse cells, thereby stimulating the re-
lease of  two types of neurotransmitters.  The  two types are glutamate
and aspartate.

Those types of neurotransmitters then stimulate receptors in the post
synaptic cells, known as N-methyl-d-aspartate receptors.  Abbreviat-
ted "NMDA receptors", they react by producing nitric oxide from their
own sites, thereby maintaining the inordinately high level of nitric oxide
already present.  Nitric oxide's ample presence proceeds to maintain the
inordinately high levels of  peroxynitrite.

While the NMDA receptors maintain an elevated nitric oxide level, per-
oxynitrite is engaged in causing the cells that contain those receptors to
be depleted of their energy pools.  Adenosine triphosphate is what's be-
ing depleted in the process.  Now, ATP is the carrier of energy in all liv-
ing organisms, and peroxynitrite inhibits mitochondrial function.  There-
for, it inhibits the production of ATP.

When cells containing NMDA receptors become deprived of their ener-
gy pool's replenishment, the NMDA receptors become hypersensitive to
stimulation.  And while the cells containing NMDA receptors are being
deprived of energy replenishment, peroxynitrite is engaged in yet another
process; that of  breaking down the blood brain barrier.  This enables in-
creased chemical access to the brain.

Meanwhile, nitric oxide performs yet another function; that of  inhibit-
ing cytochrome P450 activity.  Therefore, nitric oxide is proposed to
inhibit the process by which chemicals get metabolized and become
harmless.  The result is heightened sensitivity to chemical exposure.

The aforementioned scenario was proposed by Dr. Martin L. Pall, of
the School of  Molecular Biosciences of  Washington State.  The afore-
mentioned scenario is called "a vicious cycle mechanism"  and a paper
written by Dr. Pall which describes this vicious cycle.

http://www.allergyresearchgroup.com/Explaining-by-Martin-Pall-PhD-sp-35.html 

Vanilloid Receptor TRPV1

Recently added to this proposed mechanism is the first member of the
Vanilloid Receptor family, TRPV1.  The involvement of  TRPV1 in
MCS is the subject of a paper written by Drs. Pall and Julius Ander-
son, M.D., Ph.D., of West Hartford, Vermont;   The Vanilloid Re-
ceptor as the Putative Target of Diverse Chemicals in Multple 
Chemical Sensitivity.  The bibliographical citation for it is Arch
Environ Health. 2004 Jul;59(7):363-75.

The vanilloid receptor is implicated as a major target for a number of
chemicals which can activate it.  Therefore, vanilloid receptor activa-
tion is proposed to be the point where the vicious cycle begins.  The
vanilloid receptor paper also addresses the phenomenon of  masking,
a phenomenon duly noted in Central Chemical Sensitivity. 

    The Phenomenon of Masking is actually Dephosphorylation

Masking is the phenomenon where a chemical exposure scenario gets
muted at the outset by the overshadowing effect of  a previous and dif-
ferent one.  That same chemical exposure would have resulted in a not-
able adverse reaction if  it were the first one of that day.  The same ex-
posure will result in an adverse reaction when it becomes the first one,
on some future day.  The masking effect muted the presence of  that
specific chemical exposure for that particular day.

Masking is liken to drinking scalding coffee.  After having done so, ev-
en cold water gives a scalding effect.  Yet, if the cold water were taken
before the scalding coffee, it would have no ill effect.  Thus, after having
been exposed to one incitant (trigger), there is an inability to differentiate
between things to which you are hyper-reactive and things to which you
are not.

The authors of  the vanilloid receptor paper propose that masking occurs
during a cyclic phase known as dephosphorylation.  It's a phase triggered
by Ca2+ calmodulin phosphatease calcineurin.  Vanilloid receptor activity
is decreased during that phase; the "desensitization" phase.  Conversely,
it is during the alternate phase, the one known as phosphorylation, when
vanilloid receptor activity increases, and hypersensitivity reactions resume.
Therefore, the phosphorylation state determines the activity or inactivity
(desensitization) of  the vanilloid receptors.

In addition to the paper that Martin Pall co-authored, there is an article
on the vanilloid receptor that he individually authored.  Titled, Multiple
Chemical Sensitivity: towards the end of  controversy.  It was pub-
lished in the August/September 2005 edition of  Townsend Letter for
Doctors and Patients.  It can be accessed by clicking on the following
web address:

http://academic.research.microsoft.com/Paper/6500302

Now, the proposed mechanism of Dr. Pall is a hypothesis.  It is a hypo-
thesis which involves intricate details and intricate mapping.  This means
that the objective medical findings of chemically sensitive patients contin-
ue to carry the sole weight in proving that chemical sensitivity is a physi-
ological condition and not a psychiatric one.

The objective medical findings include instances of  anaphylaxis triggered
by nontoxic/ambient/therapeutic levels of  chemical-bearing agents.  The
findings include cases where two entirely different species of  localized
chemical sensitivity were found co-existing in the same one patient, and
such co-existence hints of the authentic existence of  MCS. 

Peripheral Chemical Sensitivity

This general type of chemical sensitivity is proposed to involve the per-
ipheral tissues.  Reactive Airways Dysfunction Syndrome is placed in
this category, as is Reactive Upper-airways Dysfunction Syndrome.
The contact sensitivity conditions, such as Airborne Irritant Contact
Dermatitis, are also placed in this category, as is Occupational Asth-
ma due to low-weight molecular agents..

This type of chemical sensitivity is proposed to involve neurogenic in
flammation.  One can obtain more information on this type of chemi-
cal sensitivity by clicking on the following links:

Hypothesis for Induction and Propagation of 
Chemical Sensitivity Based on Biopsy Studies.

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1469810/

Neurogenic Inflammation and Sensi-

tivity to Environmental Chemicals.

http://www.herc.org/news/mcsarticles/meggs-full.html
________________________________________

October 9, 2024

Corporate Welfare: Government Paying
for Illnesses Caused by Corporations

Concerning the woman whose medical records contain the following
findings, would the reasonably minded person conclude that she has
a psychosomatic illness or a physical one?

  1 - Wheezing.
  2 - Tachycardia.
  3 - Hypopotassemia.
  4 - Rales and crackles.
  5 - Gruntled breathing.
  6 - Erythematous uvula.
  7 - Grossly enlarged turbinates.
  8 - Erythema of the oropharynx.
  9 - Edema of the true vocal cords.
10 - Adenopathy in the left postauricular region.
11 - Productive response in Spiriva challenge testing.
12 - A circumscribed nodule in the left occipital region.
13 - Thickened coating over the dorsum of the tongue.
14 - A firm 1x1 cm nodule in the right postauricular region.
15+ A couple additional findings consistent with Rhinitis.

Unless you are an avowed liar, the answer to that question is beyond
obvious.  Therefore, what right do corporate-funded attorneys and an
independent medical examiner have in asserting that the woman found
to have these objectively observed ills is mentally ill?  What gives them
the right to claim that she has no objective medical findings that would
validate her symptoms?  Her symptoms have included:

[1]  a stinging tongue.
[2]  shortness of breath.
[3]  burning nasal passages.
[4]  a metallic taste in the mouth.
[5]  an adrenal-like stream throughout her solar plexus.
[6]  headaches accompanied by the bruised feeling at the
      cheekbones and temples.
[7]  ice-like numbness pervading her upper-respiratory
      tract (on specific occasion.)

Moreover, diagnoses given to her have included:

(1)  Allergic and Irritant Asthma (Reactive Airways).
(2)  Glossitis (inflammation of the tongue).
(3)  Rhinitis and Turbinate Hypertrophy.
(4)  Chemical and Irritant Sensitivities.
(5)  Reactive Hyperplasia.
____________________
____________________________
_____________________________________

Introduction

The corporation involved in the following account is one whose 2005
financial report marked its stockholder equity value at $11.2 billion.
Net tangible assets were marked at $4.2 billion.

December 2006 one-line addition/update:

Until mid-2006, the corporation involved was legally known as the
Cendant Corporation, of  Parsippany-Troy Hills, NJ.  In addition,
the geographic region involved in the following account is Elizabeth-
ton, Tennessee.  Cendant Corporation once operated business there.

The apex of  the following account concerns a year when the corpora-
tion's CEO received $17 million in salary and bonus income.   During
that same year, $1.02 million was placed into the CEO's pension fund.
An additional $4.54 million of  stockholder  money was used to pay
the premiums on his $100 million life insurance policy.

The place of work involved in the following account is a former coal tar
pitch research center.  The corporation no longer uses it.  It was found
to have minute sized monfilament fibers pervading the employees' work
areas.  And needless to say, the smaller the molecular agent, the greater
is its potential to infiltrate and afflict the complex human anatomy.  After
all, this was the case with a number of  WTC cleanup crew members
exposed to the Manhattan site's pulverized concrete dust. It resulted in
Small Airways Disease.

This account highlights a former employee of that corporation.  She
was in the process of  loosing the sum total of  everything during the
same year when the corporation's CEO was amassing a multimillion
dollar income. Throughout the account, she is simply referred to as
"the woman."  That is to say, her name will not be used.  In addition,
other persons who worked in the former research center reported
symptoms similar to hers.

The corporation was advertised as the world's largest real estate brok-
erage franchiser, the world's largest vacation ownership organization,
and the world's largest "provider of outsourced corporate employee
relocation services," as well as one of  the largest hotel franchisers in
the world and one of  the world's largest car rental operators.  Opera-
tions included the telemarketing of its services.

                      Not Even a Get Well Card

During her six months with the corporation, the woman highlighted in
this account generated approximately $500,000 in sales revenue.   In
fact, her sales of hotel room reservations averaged $2,777 per four
hour shift.  In return for her services, she was rewarded with a chron-
ic disability.  To this day, the corporation has offered her no apology,
while thecorporation's insurer has offered her no compensation.  She
was not even sent a Get Well card.  Instead, the defense attorneys and
independent medical examiner involved in her workman's comp case
sought to have her tagged with a psychiatric label which can conveni-
ently serve as an excuse for the severe illness which developed during
her time of work at the former coal tar pitch research center.

She Had to Fire Her Attorneys, in their Gross Negligence
       That is to say, she had to file a voluntary dismissal

The woman won her Social Security disability case a year ago. But, in
December 2005, she had to fire the legal counsel involved in her work-
man's comp case.  Technically speaking, she filed a voluntary dismissal.
Her attorneys refused to enter into evidence recent medical findings that
resulted from an October rhinolaryngoscopy.  And in the refusing there-
of was mention of  the cost of entering the new evidence.

The attorneys furthermore refrained from emphasizing the woman's pri-
or medical findings.  In fact, they accepted as uncontroverted truth the
averments (assertions/allegations) of  the defense counsel.  Such aver-
ments would have resulted in the woman loosing her case, and those
averments contradict her medical records.  Therefore, the woman had
to dismiss her attorneys and start anew.

HER OBJECTIVE MEDICAL FINDINGS

The defense counsel in her workman's compensation case asserted that
she had no objective medical findings to support her symptoms.  In fact,
a mental health person diagnosed her as having agoraphobia, along with
panic anxiety.  Yet, she has over a dozen objective medical  findings at-
tached to her medical records.  Such findings indicate the presence of a
physical illness, and not a psychiatric one.

Furthermore, three board certified physicians diagnosed her outside of
emergency room settings.  And those diagnoses are much different than
the one given by the "mental health person."

One of the diagnosing physicians is an allergist & immunologist, while
another one is a cytopathologist (a cytopathologist diagnoses illness at
the cellular level.) And the third diagnosing physician is an Ear, Nose,
& Throat specialist who is also a fellow of  the American College of
Surgeons.  The diagnoses given to the woman came predominately
from the fine-needle biopsy, the fiberoptic rhinolaryngoscopy, and
ER room records.  In examinations and testing performed outside of
emergency room settings, the woman was found to have:

(1)  Grossly enlarged turbinates
(2)  Erythematous uvula.
(3)  Edema of the true vocal cords.
(4)  Adenopathy in the left postauricular region.
(5)  Thickened coating over the dorsum of the tongue.
(6)  A firm 1x1 cm nodule in the right postauricular region.
(7)  A circumscribed nodule in the left occipital region.

Plus, attending ER physicians recorded the following findings:

(1)  Wheezing.
(2)  Tachycardia.
(3)  Hypopotassemia.
(4)  Gruntled breathing.
(5)  Rales and crackles.
(6)  Erythema of the oropharynx.
(7+) A couple additional findings consistent with Rhinitis.

Her diagnoses were:

(1)  Allergic and Irritant Asthma (Reactive Airways).
(2)  Glossitis (inflammation of the tongue).
(3)  Rhinitis and Turbinate Hypertrophy.
(4)  Chemical and Irritant Sensitivities.
(5)  Reactive Hyperplasia.

Upon certain environmental exposures, her symptoms reproduce them-
selves in a predictably reoccurring pattern.  Nothing about her symp-
toms is random.  In fact, due to the predictability of  her condition, she
quit keeping a diary of  her ills as far back as June 2003.  Furthermore,
she has been in need of filtered masks and air cleaners, as well as ready
access to oxygen.  In addition, prescription medications posted in her
medical records are consistent with one who has severe asthma.  Her
medications have included Albuterol,  Ipratropium Bromide, Xopenex,
Levalbuterol Hydrochloride, and Salmeterol, as well as intravenous
steroids.

Keep in mind that she was exposed to obscenely inordinate amounts
of dust at her place of work (as is described in her exposure history
account.)  Well, she tested severely positive for dust mites (in RAST
testing, I believe), while having tested negative for every other type of
high weight molecular agent (such as ragweed.)  In as much, a person
can become sensitized to dust mite proteins as much as he/she can be-
come sensitized to formaldehyde, glutaraldehyde, phthalic anhydride,
etc.  In fact, barn workers have been documented as having become
sensitized to storage mites.

HER EXPOSURE HISTORY
(transposed from her diary)

THE FIRST 3 MONTHS

April 10th 2002 Health:  Perfect
Mental Health:  "Optimistic, inspired, forward looking".

She moved from Tuscaloosa, AL to Johnson City, TN.  Jogged and
walked everyday. Could go up and down the entire complex; a span
of about four miles.  Went to the library, to Bristol Stores and malls.
Explored the local university and the book stores.  She generally did
what one does when one moves to a new city.

May 2002 Health:  Perfect

She was hired by the previously mentioned corporation, and put into
a two week training program.  Near the end of the two weeks, she de-
veloped what appeared to be the flu.  This included a sore throat and
fever, along with body aches and headaches.  She did not complete
the training at that time.

After her health improved, her training restarted.  Shortly afterward,
she was hired as a temporary and part time employee.  During the
last few days of training, one of her fellow trainees had an asthma
attack.  She noticed some stuffiness in the corridors, along with a
strange chemical odor.  But, she did not pay attention to this at the
time.

Late June/July 2002 Health:  Perfect (for the final time)

Her group was assigned to work downstairs, at the main call center.
During her first day there, she noticed a stuffy stale smell.  The chemi-
cal odor downstairs was more far intense than the smell on the floor
where she was trained.  She also noticed an inordinate amount of dust
everywhere.  Plus, on the cubicle walls were tiny and transparent fibers
the width of a human hair.  They were embedded into the cubicle walls'
fabric.  In fact, the cubicle partitions had a visible layer of brown dust
on them.  Upon a slight tap,  a cubicle wall would spew out dust.

The agent resource books were laden with dust, also.  Picking one of
them up would result in dust spewing out from the pages.  The carpet-
ing was dirty, also.  Pesticides were sprayed indoors, even with call
center employees on duty.  Some of the ceiling titles had the marks of
water damage attached to them, and within time, her fellow employees
would point out blackened mold to the woman.

THE NEXT 11 MONTHS

July/August 2002
Health:  Alternating between well and ill

She began to get a dry cough.  Things then worsened, and it became
very difficult for her to talk on the phone.  Yet, she was expected to
take a new phone call every three minutes.  She soon felt a degree of
tension in her lungs and bronchi, due to the dust and the continual talk-
ing.  She resorted to throat lozenges, Tylenol, and Robutussin.

While the HVAC system was being fixed, her crew was often told to
sit upstairs.  The chemical odor was still present upstairs, and in addi-
tion to that, free-standing fans were run at the far end of the call cen-
ter.  She preferred to sit at that end, being that the other end was an
entranceway crowded with smokers and cigarette butts.  Every time
the door opened, smoke would waft into the room.

During the hot summer months, whenever the air conditioning was not
functional, this same door would be left open.  Because of the obvious
air quality issues there, she requested to sit upstairs.  Her supervisor
agreed.  However, another supervisor spoke of having almost passed
out when training new employees upstairs.

While working downstairs, the former employee had trouble breath-
ing.  It initially started off as a “choking” episode each time the free
standing fans were turned on.  The fans were laden with dust, and
they were turned on frequently.  The blowing air would agitate the
dust in the room and propel it directly into the employees' breathing
space.  Many fellow employees began to complain about choking.

August 2002 Health:  Quite Ill

When upstairs, the sensation of burning eyes was very much prevalent,
as was the dry cough and the choking.  Dust was on the cubicle walls
upstairs, also.  The woman developed sinus congestion, a runny nose,
headaches, and a continual low grade fever.  She would arrive home
from work exceptionally fatigued.  Many of her fellow employees who
were stationed upstairs had the same symptoms.

On one occasion, as she was going upstairs to clock in, her heart be-
gan to palpitate furiously.  The staircase had not been cleaned, or if it
had been, the cleanliness had not lasted long.  Furthermore, the heavy
chemical odor was present.  In addition, there was a strong musty and
greasy smell.

Her hands began to sweat, her knees started to shake, and a tightness
in her chest was making it hard for her to take in a breath.  She was al-
so dizzy.  She went to her work area and clocked in.  She then realized
that if  she didn't get fresh air soon, she would pass out.  She went out-
side and then walked to a nearby gas station, getting a package of Ben-
adryl and something to drink.

September 4th, 2002.  First ER Visit.
Health:  Declining

She was now starting to feel fairly bad on an everyday basis.  She no-
ticed that she felt better at home.  It was only when she was at work
when her symptoms were induced.  This included the dry cough, the
burning eyes, the choking, and the palpitations that would begin soon
into the shift.  In addition, her nasal passages, throat, and lungs felt as
if they were filled with grittiness.

This was the time when she first went to an ER.  She was prescribed
Claritin and Biaxin, having been diagnosed as having Allergic Rhinitis.
The doctor noted on her records that she had a fever, rhinorrehea, and
erythema of the oropharynx, along with post nasal drip.  He also noted
abnormal constitutional signs.

She continued to treat herself with Benadryl, as it was getting progress-
ively difficult for her to work.  After twenty minutes into a work shift,
she would start coughing.  She could now hardly speak on the phone
and the Benadryl made her sleepy.  Her throat hurt and her voice now
squeaked, breaking-up frequently.  The heart palpitations continued.

A co-worker told her that he had begun to have these same types of
symptoms soon after he had started working there.  He also said that
it seemed to be getting worse for him in 2002.  Another employee told
her that he had frequent heart palpitations when at work, in addition to
the dry cough.

September 8th 2002 Second ER Visit

She began work at 8 p.m and worked until 2 am. Throughout this time
she felt a tightness forming in her chest area.  She was taking Children's
Benadryl and thought that this anti-histamine would be sufficient.  Due
to these exposures, she had a lot of congestion, along with dry cough-
ing.  She completed the shift with much difficulty.

After work, as she was driving out of the parking lot when she began to
choke.  She tried to cough but no phlegm emerged.  She pulled over at
a gas station and called Emergency Medical Services.  The EMS crew
gave her an albuterol breathing treatment in the vehicle.  She was then
taken to a hospital.  The treating physician prescribed Volmax and an
inhaler.  In fact, he stated in her medical records that she was allergic to
the work environment.  He noted the following: "Constitutional signs:  
abnormal; Tachycardia."
=================================================

October 8, 2024

The Icy Numbing

                            "Imagine a strange metallic taste and magnify it at at least 
                             50 times.  Then imagine it pervading your nose, throat,
                             larynx, tongue, bronchi, and brain  . . .  smashing you 
                             completely." 

                          "A very weird state.  Hard to explain.  Almost the feeling 
                            one gets when exposed to subzero temperatures.  Your 
                           membranes seem to get anesthetized.    Yet, they make 
                           their presences known, despite the absence of sensation."

March 2003 Health:  fair/fatigued
Emotional Outlook:  well/optimistic


She was now practicing Avoidance.  This is the practice of avoiding
the airborne agents that trigger one's asthma.  It's a practice advocat-
ed in Report 4 (A-98) of   the AMA's Counsel on Scientific Affairs.

On the woman's mind at this point in time was her plan to go to Hunts-
ville, Alabama and search for an apartment there.  This created hope-
ful optimism in her.  Job opportunities were opening in Alabama, and
a physician told her that moving away from Johnson City could reduce
her frequency of asthma.  This optimism negates any suspicion that her
ills were triggered by anxiety or depression during this time span.

March 16, 2003


She went to a grocery store, in order to buy some last minute items for
her trip.  She had been in the store for only a minute, when an asthma
attack was triggered.  After all, the store was laden with strong odors,
and the AMA has already defined strong odors as asthma triggers.  On
this occasion, her inhaler took much longer than usual to work.  On this
occasion, she became disoriented for the first time.  On this occasion,
she had entirely lost her sense of direction.  An EMS crew had to drive
her home.

March 18, 2003  EMS call - hospitalized.
Health: severe illness
Mental outlook:  scared after the attack


Being that her trip had been arranged, she convinced herself that she
could travel.  So, she and her son left for Alabama.  En route to her
destination, she suddenly became sensitive to vehicle exhaust fumes.
Her face seemed to get hot and swollen, while a gland near her ton-
sils seemed to enlarge.  Chest tightness & asthma then set in.  It felt
as if a 10 inch ball of burning fire (exhaust fumes) hit her in the chest,
spreading throughout her entire body.  She could taste the petroleum
odors.

In having become too weak to continue the trip, she searched for an en-
vironmentally friendly hotel.  Her son finally located a room that seemed
suitable for her.  Yhe result was that the irritants triggered another asth-
ma attack.  She was placed on oxygen for six hours, and given breath-
ing treatments via Xopenex, Atrovent, and Salmeterol.  She was given
the intravenous form of steroids every four hours.  That night, while in
the hospital, her blood pressure dropped drastically.

She had been diagnosed with: (a) Acute and severe asthma attack, and
(b) Hypopotassemia.  Objective medical findings already noted on re-
cord, were:   (a) labored breathing, (b) wheezing, (c) rales, (d) rapid
heart beat, (e) gruntled sounds.  This negates the defense attorneys' al-
legation of mental illness.  Then came the October 2005 rhinolaryngo-
scopy whichindicated the presence of  a physical illness much more in
depth.

March 20, 2003  Health:  debilitated.


She recalled being barely able to function in Huntsville.  Yet, she and
her son attempted to explore the city.  When waiting for a traffic light
to change, she started to become hypersensitive to exhaust fumes once
again.  She had another asthma attack.  This time, her inhaler did not
seem to help.

Wwhen driving back toward the hotel, she completely lost her sense of
direction once again. This is the second time that this happened.  EMS
personnel escorted her back to the hotel.

March 21, 2003  E.M.S call/Huntsville hospital


She went to a health food store.  Her son went inside, while she waited
outside.  He bought rosemary juice and suggested that she put some in
her bath water, thinking that a warm bath would help her.  Being that
she had always enjoyed the odor of Rosemary, she put a small capful
into the bath.  It was a mistake to have done that.  The bath was relax-
ing, but upon coming out of the tub, she starting to feel warm, and then
faint, being unable to take in a full breath.  For the third time on this trip,
she became totally confused, while feeling very lightheaded.   Her son
called EMS, and en route to the hospital, she was administered oxygen
and an IV.  At the hospital, she was given saline.

March 22, 2003  Health:  very bad; EMS call


She realized that it was not possible for her to live in Huntsville, being
that it seemed to have a lot more vehicular exhaust than did Johnson
City.  Yet, she felt that she had to complete the trip to Tuscaloosa as
planned.  She did. 

March 23, 24, 25, 2002


She spent 3 days in Tuscaloosa with friends and  had trouble breathing
outside their house as much as inside of  it.  Her friend smoked indoors,
so she stayed with her friend's sister.  She went to a Chinese restaurant
with friends, but had to leave, because of  its indoor air quality.  It was
the familiar type of irritant airspace.

She and her friends went to Books-a-Million.  She would spend many
hours there when she lived in Tuscaloosa, with no problem being there.
This time, however, she had been seated for no more than five minutes,
when the aroma of coffee became too noxious to her.  Another asthma
attack was  triggered.  So, she went outside and resorted to her inhaler.

Late that night, her friend's sister turned on a gas heater.  An ensuing
odor made the woman sick, bringing her close to having another asth-
ma attack.  She had to sleep next to a window, while wearing a filter
mask throughout the entire night.  Her friends then took her to church
the next day, and she sat next to the open back door, wearing two fil-
ter masks.

She went back to Johnson City that week.  On the way back, she got
stuck in traffic.  She became ill once again.  When she finally reached
home, she collapsed out of weakness.   She also had a headache, a
sore throat, and chest inflammation.  This collapse mode lasted for the
next few years.

Since April, 2003, she had varying degrees of tightness in the chest,
along with asthma, nasal pain, burning, and congestion.  She took all
of her prescribed medications, and resorted to wearing activated car-
bon masks much of  the time.  In addition, she has a car interior air
cleaner, as well as dust screens for the car.  This helps, but she is
still able to smell odors, even through the carbon masks.

There were two doctor's appointments that she was unable to keep.
One was missed because she couldn’t find the doctor’s office in the
midst of another irritant response to exhaust fumes.  The other physi-
cian had his office in a commercial building downtown.  As she was
approaching the entrance, she caught sight of nurses smoking.  She
knew that she would not be able to make it down the smokey, fra-
grance laden, and cleaning agent laden corridor.  Her need to find a
primary care physician was pressing.

May 31st, 2003  Health:  Depends on exposures.
Mental:  Good/Fair


She found two doctors at a nearby university who understand her type
of medical condition.  They did all they could to help her.  She was ap-
prehensive on her first visit to one of the newly located physicians, and
it was with the utmost self-control that she waited in the waiting room.
This was due to her extreme sensitivity to fragrances and various clean-
ing agents.  Blood tests were ordered after a lengthy consultation.  Yet,
en route to the testing  area, she started to feel ill again, more so than
previously.

She was once scheduled to take a CT scan.  The building where the
scan was to be taken was a bit worse than the building where she had
previously been.  The technician had the woman go into the machine
promptly and performed the scan.  Meanwhile, she began to feel light
headed.  It seemed as if a metallic smell were causing it.  In fact, she
had to be helped out of  the room, after the scan.  She felt dizzy, even
to the point where she felt that she was about to pass out.

She remained ill up to 48 hours after the CT scan.  After the scan, she
was short of  breath.  But, this form of  shortness of breath was much
different than the previous bouts.  It appeared to be a tissue reaction,
she said; one accompanied by a hollow lack of sensation and even a
numbness.  This absence of sensation extended to her  nose, bronchi,
esophagus, trachea, and lungs.  She said that her lungs hurt.  But, it was
a dull chronic inflammation that she felt.  She described it in the follow-
ing manner:

      "A very weird state.  Hard to explain.  Almost the feeling
      feeling one gets when exposed to subzero temperatures.
      Your membranes seem to get anesthesized.  Yet, they
      make their existences known, despite the absence of
      sensation."

   "All this is very strange.  It's anxiety-causing, because if it's

     not ameliorated, it leads to a strange sort of  “inability" to 
     breath.  It's not like congestion, in the usual sense.  It leads 
     to the desire to cough, but the cough does not lead to any
     cessation of  symptoms.  I also feel as if I'm in the process 
     of fainting at times.  I feel shakey."

June 05, 2003

She began to feel better, two days after the CT scan.  The “hollow” 
and metallic” syndrome finally resolved itself.  About this she wrote:
"Just that little exposure to the CT Scan environment made me 
ill for two days!"

She had to go to the bank one day, and it was a hot and/or humid 84
degrees outside.  She started to feel ill in the heat, with the activated
carbon mask on.  The mask itself was starting to emit traces of ex-
haust odor.  She never made it to the bank that day.
==============================================

October 7, 2024

An Examation Report

The Chemically Sensitive Woman with More
Than a Dozen Objective Medical Findings:

Let us review.  The stereotypical pro-chemical and pro-corporate
propaganda has repeatedly been that chemically sensitive persons
have no objective medical findings to validate their symptoms, de-
spite the fact that RAST TESTING includes tests for chemical al-
lergies ... despite the recognition of Reactive Airways Dysfunction
Syndrome ... despite the fact that the golden rule for diagnosing
Irritant-associated Vocal Cord Dysfunction is the very objective 
Fiberoptic Rhinolaryngoscopy ... and despite the existence of the 
diagnosis of Occupational Asthma due to Low Weight Molecular 
Agents.

Posted below is one of  the pages in the medical record of  the pa-
tient high-lighted in The Chemically Sensitive Woman Who Has
More Than a Dozen Objective Medical Findings.  The report
records grossly enlarged turbinates, shoddy adenopathy, and a 
thickened coating over the dorsum of  tongue, among other things.
Quite frankly,  the grossly enlarged turbinates were enough to illu-
strate the presence of  a physical illness.   In the total tally, her med
records report her as having:

- Wheezing.
- Tachycardia.
- Hypopotassemia.
- Gruntled breathing.
- Rales and crackles.
- Erythematous uvula.
- Blistering of the tongue.
- Grossly enlarged turbinates.
- Erythema of the oropharynx.
- Edema of the true vocal cords.
- Adenopathy in the left postauricular region.
- Thickened coating over the dorsum of the tongue.
- Productive response in Spiriva challenge testing.
- A circumscribed nodule in the left occipital region.
- A firm 1x1 cm nodule in the right postauricular region.
- A couple additional findings consistent with Rhinitis.

The bottom line concerning this woman is that, whatever be her ill- 
ness, it's one of  a physical nature and not a matter of  psychiatric
illness.   The more universal bottom line is that there are chemically
sensitive people who have objective medical findings that validate
their sufferings.  In all such cases, allegations of  mental illness con-
stitute a defamation of  character hurled against an entire class of
people.   Clicking on the photo below will enable you to read the
medical report.  In addition, Glossitis mentioned in the following 
report is inflammation of the tongue.